Sleep Apnea And The Heart And Stroke Connection

Sleep Apnea And The Heart And Stroke Connection

What do I need to know about the relationship between sleep and AFib?

Plain old snoring can get a little annoying, especially for someone listening to it. But when a snorer repeatedly stops breathing for brief moments, it can be worrisome for those listening and it may also provide an important clue about a problem that should be addressed.

Not everyone who has sleep apnea snores, so a person’s nighttime breathing habits may go unnoticed, however quality sleep is very important for heart health, and many people know very little about the quality of their sleep. The evidence is very strong for the relationship between sleep apnea and atrial fibrillation as well as an increased risk for hypertension and cardiovascular disease.

What is sleep apnea?

Imagine waking up choking and gasping for air, then catching your breath and falling back into fitful slumber – for a minute or two, until it happens again. And again. And again. Five to thirty times a night.

During an episode of sleep apnea, a person may experience pauses in breathing five to 30 times per hour or more during sleep. These episodes typically wake or startle the sleeper when he or she gasps for air, although the sleeper may never remember these struggles to breathe. Even so, it prevents restful sleep and is associated with high blood pressure, arrhythmias like AFib, stroke and heart failure.

Sleep Apnea: A Common Problem

  • Sleep apnea is commonly seen with AF.
  • People who have moderate-to-severe sleep apnea are approximately 4 times more likely to have AFib than those who do not have sleep apnea.
  • The occurrence of apneas during sleep may serve as triggers to episodes of AFib.
  • Sleep apnea is more common today and those who are overweight are more likely to be at increased risk for sleep apnea as well as AFib.
  • AFib patients with untreated sleep apnea may be more likely to revert back into afib after electrical cardioversion than other afib patients without sleep apnea.
  • For those who have AFib, treating the sleep apnea may improve the atrial fibrillation.
  • One in five adults suffers from at least mild sleep apnea, and it afflicts more men than women.
  • The most common type is obstructive sleep apnea (or OSA) in which the flow of air is blocked, sometimes attributed in part to excess body weight that causes the tissues in throat to collapse, blocking the airway.
  • The other types are: central sleep apnea (CSA) and mixed apnea, which is a combination of the other two types.

Why is there a correlation between sleep apnea and heart disease?

  • Sleep apnea, heart disease, and obesity are all linked with AFib.
  • Any type of airway obstruction increases risks. Anyone who is overweight, has large tonsils or adenoids, suffers chronic nasal congestion, or has other structural abnormalities blocking the airway has an increased risk.
  • Some people are more likely to develop sleep apnea. The most common scenario would be a man over the age of 40 who is overweight and can't button the top of his shirt collar, unless it is 17 inches or larger, but OSA can affect women or children, as well.
  • Sleep apnea and blood pressure are related. Up to half of people who have OSA also have high blood pressure-and unlike most people, their blood pressure does not fall while they're asleep. Essentially, their hearts do not get the same benefit from rest.
  • Lack of oxygen and a failure to fully rest are probable causes for increased blood pressure. Scientists aren’t sure whether the repeated awakenings from sleep apnea elevates blood pressure or if blood pressure rises because the oxygen levels in the blood are reduced. It may also come from a combination of both.
    High blood pressure increases the risks for stroke for all people, including those people with AFib who are already at increased risk.
  • About half of AFib patients have obstructive sleep apnea, a stronger correlation than between AFib and any other risk factors.
  • Chronic inflammation is correlated with sleep apnea and is suspected to be a factor for many people with AFib. High levels of C-reactive protein (CRP), a marker of inflammation, indicates the possible presence of heart disease and may exacerbate existing diseases of the circulatory system.
  • With CSA, airway blockage isn't the problem. It's that the brain doesn't send regular, continuous signals to the muscles of the diaphragm to contract and expand to enable you to inhale and exhale. Central sleep apnea may develop after a stroke, or may be caused by sedatives and narcotics and brain injury, explains Undevia. Heart failure patients are susceptible to developing a type of CSA called Cheyne-Stokes respiration.

How would I know if I have sleep apnea?

  • Listen to those snoring comments.
    • Has your spouse delicately suggested separate bedrooms because your snoring has become unbearable?
    • Has a roommate or family member ever commented that they thought you might not be breathing or that you suddenly began to gasp during your sleep? Often sleep apnea is first noticed by a roommate or sleeping partner of someone with sleep apnea. It can go unnoticed in a person who lives alone, unless a sleep study is ordered or requested.
  • Notice your level of fatigue and concentration.
    • Do you often feel sleepy despite being in bed eight hours a night?
    • Are you prone to accidents or do you fall asleep during the day?
    • Do you feel unable to function unless you have a steady stream of caffeine throughout your day?
  • In addition, consider tracking your sleep during the night for an informal self-study.
    • Although it’s no substitute for a medical sleep study, recording yourself may help you identify some specifics to discuss with your doctor.
    • Several smartphones and computer apps offer inexpensive ways to monitor your sleep and even your corresponding heart rates. Sharing what you learn with your healthcare provider can help make the case for support if a sleep study is warranted.

If you answered yes to the above, talk with your doctor or healthcare provider about doing a sleep study.

  • In addition, consider tracking your sleep during the night for an informal self-study.
    • Although it’s no substitute for a medical sleep study, recording yourself may help you identify some specifics to discuss with your doctor.
    • Several smartphones and computer apps offer inexpensive ways to monitor your sleep and even your corresponding heart rates. Sharing what you learn with your healthcare provider can help make the case for support if a sleep study is warranted.

Getting good rest is important for your heart.

Whether or not you’re in need of a sleep study, if you’re struggling to get a good night’s sleep follow some of these suggestions:

  • Get regular physical activity, but don’t do it right before bed because that gets your adrenaline pumping and can keep you awake.
  • Limit alcohol consumption to one drink per day for women and two drinks for men; too much alcohol interferes with sleep.
  • Avoid caffeine before bed.
  • Develop a pre-bedtime routine such as taking a warm bath, dimming the lights or having some herbal tea.

What is a sleep study?

You will spend a night or two in a lab hooked up to a machine that will monitor and chart your brain waves, oxygen levels, breathing and heart rate while you sleep.

The brain-wave pattern indicates whether you are asleep, and the quality of your sleep. You will be monitored for pauses in breathing, and whether you wake up during those pauses. During breathing pauses, oxygen levels are checked, and your heart rhythm is tracked to find out whether heart rate slows or becomes irregular.

Getting a diagnosis

Generally, a diagnosis is based on the number of pauses in breathing each hour.

  • 5-15 pauses in breathing an hour may be a mild case of apnea
  • 15-30 may fall in the moderate range.
  • 30+ may indicate a more severe case.

If the test shows that you have sleep apnea, certain lifestyle changes might be recommended, but you will also most likely be fitted for a continuous positive airway pressure (CPAP) or other doctor-recommended airway device while you sleep, or possibly a mouthguard if you have just mild sleep apnea.

How is sleep apnea treated?

  • You may consider using a CPAP while you sleep or other doctor-recommended airway device while you sleep.
    • As you inhale, an air compressor in the device creates enough pressure to produce a steady stream of air that keeps your airway open.
    • As an added bonus, some studies show that elevated blood pressure drops significantly after using the mask for two weeks.
    • Although the mask is effective, some patients complain of claustrophobia, or have trouble falling asleep while wearing it. Trying a mask with a different style or a different fit may solve the problem, or a mouthguard may work if you have mild sleep apnea.
    • Newer options are in development that work on the central nervous system, so ask your healthcare provider to see if any of these new treatment choices may be right for you.
  • Some opt for surgical treatment.
    • When someone with a case of sleep apnea is resistant to using the mask, there are surgical alternatives.
    • Changing the shape of your airways: Surgical procedures can make the back of the throat larger, provide support so tissues in the roof of the mouth no longer collapse, or reduce the size of tissues in the nose.
    • Using a thin, lighted scope snaked up your nose, an ear, nose and throat (ENT) surgeon will see where the blockage is, says Undevia, and then may recommend having your tonsils removed or correcting a deviated septum in the nose.
    • Check with your healthcare provider to learn more about specific options that may work for you.

Forcing the airways to remain open: Doctors used to alleviate sleep apnea by performing a tracheostomy – cutting a hole in the windpipe – which is still an option in severe cases when CPAP does not work.

Sleep apnea isn't just snoring. Left untreated, the condition can lead to serious cardiovascular problems. Fortunately, there are several non-surgical and surgical treatment options you can discuss with your doctor.

Recent Discussions From The At Home Forum
barbara1642 avatar

Do patients with a-fib ever get pacemakers?  I haven't posted lately because I've been fine.......and having other issues:  dental & cataracts.  The joys of getting   

So do we fibbers ever get pacemakers?  2 days ago I had an "incident", and I think my heart wants to go into a-fib, but it doesn't quite make it.  I have an appt w/my cardio this morning.   


I'm just curious..............


live long & prosper

Barbi1 avatar

hi I’m new , I was diagnosed with Ischemic in May , had surgery 8 May I went AFIB on the table , I hope I’m saying this right .  I’m on xarelto . I need advice on eating right .  Thanks Barbi 

maria12045 avatar

This concerns my father.

Age: 62, caucasian male

Location: Greece

Weight: 71kg

Height: approximately 175cm

Diagnosis: dilated cardiomyopathy and atrial fibrillation (he is currently not in a-fib: his atrial fibrillation stopped after ablation)

Other conditions: none besides retinal detachment in one eye, and raised intraocular pressure in the other which he treats with eyedrops; and two instances of bells palsy in the past.

Current prescription:

Xarelto 50mg (rivaroxaban)

Entresto 100mg (sacubitril/valsartan)

Carvepen 25mg (carvedilol)

Eleveon 50mg (eplerenone)

He also took Zyloric 100mg (allopurinol) to manage uric levels which he quit along with the above.


September 2016 my dad went to ER with chest/abdominal pain. They found he had EF of 24% and left ventricle 60mm.

His latest check up, April 2019 he had a cardiac ultrasound showing EF of 55-60%. Left ventricle approx 53mm.

On June 7, 2019 (three weeks ago now) he stopped all meds without telling anyone -not doctors or family- because he feels he doesn‘t need them. He had back pain, joint pain, fatigue and complained of insomnia and cited these as his reasons for stopping. However, I recall him beginning questioning the necessity of meds as soon as he got rid of the AF by ablation. He has been reading studies about the long term side effects of heart medications and this motivated him to quit as well.

Since finding out three days ago, myself, family and his doctors have advised him to restart but he is ignoring this advice because he says he feels great since the side effect symptoms (back pain, fatigue, etc.) have stopped and he is checking his blood pressure regularly as a safety measure.

As to why he would doubt the medicines when it has been demonstrated that they work, I believe the answer is the following: since the beginning, his doctors described the cause of his condition as a chicken and egg situation (did heart enlargement cause the atrial fibrillation or vice versa?). I believe my dad started to attribute the cardiomyopathy to the a-fib. Therefore, since he is currently physiological (despite the fact that this a result of therapy) he thinks he only has to watch out for another episode of a-fib.. He thinks the medications have already done their job and are now just damaging his health unnecessarily. But he needs to be explained that they still are doing their job. And that the risks outweigh the side effects.

I am hoping for information about how the medicines are still contributing to his heart. And about the risks of stopping. But any feedback at all from patients, family of patients as well as medical professionals would be appreciated so much as I am at a loss.

Thank you for very much for reading.

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