Sleep Apnea And The Heart And Stroke Connection

Sleep Apnea And The Heart And Stroke Connection

What do I need to know about the relationship between sleep and AFib?

Plain old snoring can get a little annoying, especially for someone listening to it. But when a snorer repeatedly stops breathing for brief moments, it can be worrisome for those listening and it may also provide an important clue about a problem that should be addressed.

Not everyone who has sleep apnea snores, so a person’s nighttime breathing habits may go unnoticed, however quality sleep is very important for heart health, and many people know very little about the quality of their sleep. The evidence is very strong for the relationship between sleep apnea and atrial fibrillation as well as an increased risk for hypertension and cardiovascular disease.

What is sleep apnea?

Imagine waking up choking and gasping for air, then catching your breath and falling back into fitful slumber – for a minute or two, until it happens again. And again. And again. Five to thirty times a night.

During an episode of sleep apnea, a person may experience pauses in breathing five to 30 times per hour or more during sleep. These episodes typically wake or startle the sleeper when he or she gasps for air, although the sleeper may never remember these struggles to breathe. Even so, it prevents restful sleep and is associated with high blood pressure, arrhythmias like AFib, stroke and heart failure.

Sleep Apnea: A Common Problem

  • Sleep apnea is commonly seen with AF.
  • People who have moderate-to-severe sleep apnea are approximately 4 times more likely to have AFib than those who do not have sleep apnea.
  • The occurrence of apneas during sleep may serve as triggers to episodes of AFib.
  • Sleep apnea is more common today and those who are overweight are more likely to be at increased risk for sleep apnea as well as AFib.
  • AFib patients with untreated sleep apnea may be more likely to revert back into afib after electrical cardioversion than other afib patients without sleep apnea.
  • For those who have AFib, treating the sleep apnea may improve the atrial fibrillation.
  • One in five adults suffers from at least mild sleep apnea, and it afflicts more men than women.
  • The most common type is obstructive sleep apnea (or OSA) in which the flow of air is blocked, sometimes attributed in part to excess body weight that causes the tissues in throat to collapse, blocking the airway.
  • The other types are: central sleep apnea (CSA) and mixed apnea, which is a combination of the other two types.

Why is there a correlation between sleep apnea and heart disease?

  • Sleep apnea, heart disease, and obesity are all linked with AFib.
  • Any type of airway obstruction increases risks. Anyone who is overweight, has large tonsils or adenoids, suffers chronic nasal congestion, or has other structural abnormalities blocking the airway has an increased risk.
  • Some people are more likely to develop sleep apnea. The most common scenario would be a man over the age of 40 who is overweight and can't button the top of his shirt collar, unless it is 17 inches or larger, but OSA can affect women or children, as well.
  • Sleep apnea and blood pressure are related. Up to half of people who have OSA also have high blood pressure-and unlike most people, their blood pressure does not fall while they're asleep. Essentially, their hearts do not get the same benefit from rest.
  • Lack of oxygen and a failure to fully rest are probable causes for increased blood pressure. Scientists aren’t sure whether the repeated awakenings from sleep apnea elevates blood pressure or if blood pressure rises because the oxygen levels in the blood are reduced. It may also come from a combination of both.
    High blood pressure increases the risks for stroke for all people, including those people with AFib who are already at increased risk.
  • About half of AFib patients have obstructive sleep apnea, a stronger correlation than between AFib and any other risk factors.
  • Chronic inflammation is correlated with sleep apnea and is suspected to be a factor for many people with AFib. High levels of C-reactive protein (CRP), a marker of inflammation, indicates the possible presence of heart disease and may exacerbate existing diseases of the circulatory system.
  • With CSA, airway blockage isn't the problem. It's that the brain doesn't send regular, continuous signals to the muscles of the diaphragm to contract and expand to enable you to inhale and exhale. Central sleep apnea may develop after a stroke, or may be caused by sedatives and narcotics and brain injury, explains Undevia. Heart failure patients are susceptible to developing a type of CSA called Cheyne-Stokes respiration.

How would I know if I have sleep apnea?

  • Listen to those snoring comments.
    • Has your spouse delicately suggested separate bedrooms because your snoring has become unbearable?
    • Has a roommate or family member ever commented that they thought you might not be breathing or that you suddenly began to gasp during your sleep? Often sleep apnea is first noticed by a roommate or sleeping partner of someone with sleep apnea. It can go unnoticed in a person who lives alone, unless a sleep study is ordered or requested.
  • Notice your level of fatigue and concentration.
    • Do you often feel sleepy despite being in bed eight hours a night?
    • Are you prone to accidents or do you fall asleep during the day?
    • Do you feel unable to function unless you have a steady stream of caffeine throughout your day?
  • In addition, consider tracking your sleep during the night for an informal self-study.
    • Although it’s no substitute for a medical sleep study, recording yourself may help you identify some specifics to discuss with your doctor.
    • Several smartphones and computer apps offer inexpensive ways to monitor your sleep and even your corresponding heart rates. Sharing what you learn with your healthcare provider can help make the case for support if a sleep study is warranted.

If you answered yes to the above, talk with your doctor or healthcare provider about doing a sleep study.

  • In addition, consider tracking your sleep during the night for an informal self-study.
    • Although it’s no substitute for a medical sleep study, recording yourself may help you identify some specifics to discuss with your doctor.
    • Several smartphones and computer apps offer inexpensive ways to monitor your sleep and even your corresponding heart rates. Sharing what you learn with your healthcare provider can help make the case for support if a sleep study is warranted.

Getting good rest is important for your heart.

Whether or not you’re in need of a sleep study, if you’re struggling to get a good night’s sleep follow some of these suggestions:

  • Get regular physical activity, but don’t do it right before bed because that gets your adrenaline pumping and can keep you awake.
  • Limit alcohol consumption to one drink per day for women and two drinks for men; too much alcohol interferes with sleep.
  • Avoid caffeine before bed.
  • Develop a pre-bedtime routine such as taking a warm bath, dimming the lights or having some herbal tea.

What is a sleep study?

You will spend a night or two in a lab hooked up to a machine that will monitor and chart your brain waves, oxygen levels, breathing and heart rate while you sleep.

The brain-wave pattern indicates whether you are asleep, and the quality of your sleep. You will be monitored for pauses in breathing, and whether you wake up during those pauses. During breathing pauses, oxygen levels are checked, and your heart rhythm is tracked to find out whether heart rate slows or becomes irregular.

Getting a diagnosis

Generally, a diagnosis is based on the number of pauses in breathing each hour.

  • 5-15 pauses in breathing an hour may be a mild case of apnea
  • 15-30 may fall in the moderate range.
  • 30+ may indicate a more severe case.

If the test shows that you have sleep apnea, certain lifestyle changes might be recommended, but you will also most likely be fitted for a continuous positive airway pressure (CPAP) or other doctor-recommended airway device while you sleep, or possibly a mouthguard if you have just mild sleep apnea.

How is sleep apnea treated?

  • You may consider using a CPAP while you sleep or other doctor-recommended airway device while you sleep.
    • As you inhale, an air compressor in the device creates enough pressure to produce a steady stream of air that keeps your airway open.
    • As an added bonus, some studies show that elevated blood pressure drops significantly after using the mask for two weeks.
    • Although the mask is effective, some patients complain of claustrophobia, or have trouble falling asleep while wearing it. Trying a mask with a different style or a different fit may solve the problem, or a mouthguard may work if you have mild sleep apnea.
    • Newer options are in development that work on the central nervous system, so ask your healthcare provider to see if any of these new treatment choices may be right for you.
  • Some opt for surgical treatment.
    • When someone with a case of sleep apnea is resistant to using the mask, there are surgical alternatives.
    • Changing the shape of your airways: Surgical procedures can make the back of the throat larger, provide support so tissues in the roof of the mouth no longer collapse, or reduce the size of tissues in the nose.
    • Using a thin, lighted scope snaked up your nose, an ear, nose and throat (ENT) surgeon will see where the blockage is, says Undevia, and then may recommend having your tonsils removed or correcting a deviated septum in the nose.
    • Check with your healthcare provider to learn more about specific options that may work for you.

Forcing the airways to remain open: Doctors used to alleviate sleep apnea by performing a tracheostomy – cutting a hole in the windpipe – which is still an option in severe cases when CPAP does not work.

Sleep apnea isn't just snoring. Left untreated, the condition can lead to serious cardiovascular problems. Fortunately, there are several non-surgical and surgical treatment options you can discuss with your doctor.

Recent Discussions From The At Home Forum
outofafib avatar

I am a new writer here so greetings all. My Afib attack was of the stealth variety. I was not aware of anything until my stomach began to bother me. This was approximately five weeks before I landed in the hospital. It was on and off with some bouts of constipation. The condition began to worsen. The feeling was as if a hand was squeezing my stomach. I wondered if I had ulcers or something wrong with my intestines. I made an appointment to see a gastroenterologist. I never made it. The constipation became so bad I went to an emergency room to get medicinal relief. While there I took off my shoe and showed the doctor my right foot. It was swollen. Both feet were swollen. That was that. I was hooked up every heart gizmo in the room. I was informed that my ejection fraction and spiked downward to an eight and then bounced up to a fourteen. My official reading once I was in the cardiac ward clocked me at eighteen. My heart had been hammered so badly that I was in Congestive Heart Failure.  I was informed that my heart tissue was thin. My heart had dramatically increased in size. The expectation was that I would need electronics and a heart transplant. I was informed I would need to live in an assisted living facility. This was a lot of grim news coming in fast.

I had all the drugs that are used to treat someone with my condition. I did have one catheter ablation.

As I was in the hospital events gradually turned in my favor. I have been a gym ****** most of my life. My diet and nutritonal supplements follow that kind of habit. I commented on that to the nurse on my case. She stated that may be why I was as I was. She stated that others on the ward at the same level as me could not walk or talk.

I was released from the hospital under my own steam. At that point I went off script. I continued to use the traditional meds but studied nutritional supplements specific to heart and brain health. I used extreme doses. When I had my second heart scan fourt months after my ablation my heart ejection fraction was clocked at sixty-five.  I had made it. No electronics, no heart transplant, heart tissue and size in normal states. I have been able to successfuly maintain my state up to present time. I have not informed my cardiologists of what I did in the shadows. They know I did something. One of my cardiologists told me I was an odd patient. I heard a lot of Wow, Wow, Wow over and over.

This battle took place from 2012 until the winter of 2017. I was fifty-seven and as of this writing I am sixty-five. No more heart meds. No more Afib episodes. I still have to monitor my blood pressure. This is responding to exercise and nutritional adjustments.

I really do exist. Take care all.



Tmariez avatar

Hi everyone!  I had my first ablation on Friday October 30th.  I was nervous going into it but everything was over quickly and went smoothly.  My recovery was not fun but it could have been worse.  I had to lie still for 6 hours, which I know is a common thing.  My back wasn't too happy though 😊 Anyways, I stayed overnight to make sure everything was good and I was discharged in the morning.  I'm now home taking it easy.  I'm taking a couple of days off from work but I'll prob take rest of week off since I'm not in rush to go back 😊. Thanks for the encouragement and making me feel at ease of my decision.

quilabell avatar

Hi all!

I'm post-op day 5 from my first ablation (PVI cryotherapy). Although it has been a rocky road, two things are really concerning me and I would appreciate any input from you.

First, My fatigue is crazy. I feel like I have the flu without flu symptoms. I can only do a few of my ADL's, then pretty much need to lay down again. Is this usual and if so,how long until it improves? Ugh. I feel like I'm 95.

Second, I've had pretty awful headaches since I woke up from the surgery. I do have a long history of daily tension headaches, and these are the same except they do not respond to medication. I now have scintillating scotomas daily and the pain is more severe. I do not have any neurological symptoms, although I did have left hand and left cheek numbness for a few minutes (no motor defecits) that resolved when the scotoma resolved today. The headache pain is bi-temporal, throbbing and radiates to my teeth. Has anyone experienced these symptoms or should I go in? The LAST thing I want to see is a medical facility right now.

Incidentally, I am an Emergency Medicine/Trauma PA-C(Physician Assistant) with 21 years experience, 49 years old, no co-morbidities, paroxysmal a.fib x 5 years (one episode each 9 months until recently when all hell broke loose), history of Rheumatic fever and recent valve repair last month.

And no, I don't want to consult one of the many docs that I work with because this is not their specialty and they are all convinced that I am dying, which equals huge and unnecessary work ups.

Thanks in advance for any input!


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